Phenotype Details
Phene ID
4970
Name
Persistent truncus arteriosus, GATA6-related
Phene Name
N/A
OMIA ID
2559
Species ID
9913
Characterised
Yes
Characterised Year
2022
Linked Variants
Variant IDPhenotypeGene IDDeleteriousChromosomeGenomicTranscriptProtein
1501Persistent truncus arteriosus388314538Unknown24g.34187181T>Ac.1249A>Tp.(K417X)
Linked Breeds
BreedBreed IDSpecies IDVBO Term
Holstein Friesian (Cattle)739913http://purl.obolibrary.org/obo/VBO_0000239
Summary

Besnard et al. (2023) monitored “sire-family calf mortality within the French and Walloon Holstein populations, and ... [used] this information to detect genetic defects that might have been overlooked by lack of specific symptoms. … After outlining the 5 worst bulls per category, [the authors] ... paid particular attention to the bulls Mo and Pa, because they were half-brothers. Using a battery of approaches, including necropsies, karyotyping, genetic mapping, and whole-genome sequencing, [the authors] ... described 2 new independent genetic defects in their progeny and their molecular etiology. … Pa was found to be mosaic for a dominant de novo nonsense mutation of GATA 6 binding protein (GATA6), causing severe cardiac malformations.”

Molecular Genetics

Whole genome sequencing of an affected animal and comparison to control animals identified "a thymine-to-adenine substitution in exon 2 of GATA6 predicted to introduce a premature stop codon (chr24: g.34,187,181T > A; GATA6 p.K417X)" as likely causal variant (Besnard et al., 2023). The variant was validated in additional animals and "2 orthologous human truncating mutations located closest to the present bovine nonsense variant (pS418fs and pG441X) have been reported to cause exactly the same phenotype" (Besnard et al.; 2023).

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